Ncam binding

A peptide mimetic targeting trans-homophilic NCAM binding sites promotes spatial learning and neural plasticity in the hippocampus. Combinatorial transinteraction studies with cells and membranes with different. Homophilic Binding Mediated by the Neural Cell.

Demonstration of an Extracellular ATP-Binding Site in NCAM. Mechanism of Homophilic Binding Mediated by the Neural Cell. The Homophilic Binding Site of the Neural Cell Adhesion Molecule. PSA and NCAM phenotypes revealed that heterophilic NCAM binding mimics the.

NCAM as a Novel GDNF Family Receptor: R. A peptide mimetic targeting trans-homophilic NCAM binding sites. Construction, expression and binding specificity of bispecific CD3.

Polysialic acid controls NCAM signals at cellcell contacts to. Structural biology of NCAM homophilic binding and activation of. Moreover, peptide Pstimulated a 2-fold increase in neurite-bearing cells, suggesting that. PSA is thought to decrease NCAM homophilic binding (NCAMNCAM binding) and thereby attenuate cellcell adhesion (Sadoul et al.

Neural cell adhesion molecule - , the free encyclopedia Neural cell adhesion molecule (NCAM also called CD5 is a homophilic binding glycoprotein expressed on the surface of neurons, glia, skeletal muscle and). The presence and absence of polysialic acid on. Inactivation of the enzyme and affinity labeling of NCAM. In contrast, C3d had no effect on neurite outgrowth. A Peptide Mimetic Targeting Trans-Homophilic NCAM Binding Sites.

The neural cell adhesion molecule (N-CAM) plays an integral role in cell interactions during neural development, with the binding of. A new approach to identify cytosolic NCAM binding partners. An upstream regulatory element of the NCAM promoter contains a. The neural cell adhesion molecule (NCAM ) plays an important role during brain. An upstream regulatory element of the NCAM promoter contains a binding site for homeodomains.
The neuritogenic response mediated by homophilic NCAM binding was blocked by the C3d peptide.

Thus, NCAM contains an ATP-binding site, and this site is localized extracellularly and probably has the. Identification of a heparin binding domain of the neural cell. NCAM -Induced Neurite Outgrowth Depends on Binding of.

Identification of a neuritogenic ligand of the neural cell adhesion. Identification of novel cytosolic binding partners of the neural cell. Polysialic Acid Directs Tumor Cell Growth by Controlling. Reactome FGFR ligand binding and activation FGFR is probably activated by NCAM very differently from the way by which it is activated by FGFs, reflecting the different conditions for NCAM -FGFR and. Binding Properties of Detergent-solubilized NCAM The only detectable NCAM -binding proteins were identified as the 140- and 180- kD forms of NCAM.

Fragments as well as neurite outgrowth are abolished by mutation of the calmodulin binding motif in the intracellular domain of NCAM that is. NCAM -NCAM binding, induced the sprouting of mul- tiple neurites. Polysialic acid (polySia) is a major determinant of NCAM binding but also a general modulator of cellcell interactions (Rutishauser, 20Hildebrandt et al).

Recently, a decapeptide ligand of NCAM plannexin, which mimics a homophilic trans-binding site in Igand binds to Ig3was developed as. Zippers Make Signals: NCAM -mediated Molecular Interactions and. The neural cell adhesion molecule, NCAM, is involved in multiple cis- and trans-homophilic interactions (NCAM binding to NCAM ) thereby facilitating. However, the diabody derived from the murine anti-NCAM scFv could not efficiently be expressed in E. 2IUG: Crystal Structure of the Pi 3-Kinase PAmino-Terminal ShDomain and its Phosphopeptide Complexes.

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